Antiinflammatory activity in mouse J774.A1 cells assessed as decrease in LPS-induced IL-6 secretion at 20 uM preincubated for 30 mins followed by LPS stimulation and measured after 24 hrs by ELISA
Antiinflammatory activity in mouse J774.A1 cells assessed as decrease in LPS-induced TNF-alpha secretion at 20 uM preincubated for 30 mins followed by LPS stimulation and measured after 24 hrs by ELISA
Antiinflammatory activity in mouse J774.A1 cells assessed as decrease in LPS-induced IL-6 secretion preincubated for 30 mins followed by LPS stimulation and measured after 24 hrs by ELISA
Antiinflammatory activity in mouse J774.A1 cells assessed as decrease in LPS-induced TNF-alpha secretion preincubated for 30 mins followed by LPS stimulation and measured after 24 hrs by ELISA
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in neutrophil count in BALF at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by Wright and Giemsa staining based analysis
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in TNF-alpha production in BALF at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in IL-6 production in BALF at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in TNF-alpha production in serum at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in IL-6 production in serum at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in alveolar septa thickness at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by H and E staining based assay
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in pulmonary congestion at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by H and E staining based assay
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in inflammatory infiltration at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by H and E staining based assay
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in tissue destruction at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by H and E staining based assay
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in neutrophil infiltration at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by F4/80 antibody staining based assay
Antiinflammatory activity in C57BL/6J mouse model of LPS-induced acute lung injury assessed as reduction in p-p65 level at 20 mg/kg, po administered 30 mins prior to LPS treatment and measured after 6 hrs by immunohistochemical analysis