# | Aladdin ID | Assay Type | Description | Organism | Compounds | Reference | BAO Format | Source | |
---|---|---|---|---|---|---|---|---|---|
1. | ALA3800919 | Binding | Inhibition of porcine pancreatic lipase using p-nitrophenyl butyrate as substrate assessed as formation of p-nitrophenol preincubated for 10 mins followed by substrate addition by spectrophotometric method | Sus scrofa | 10 | ALA3797192 | single protein format | Scientific Literature | |
2. | ALA1041807 | Binding | Inhibition of human recombinant PTP1B | Homo sapiens | 8 | ALA1156122 | single protein format | Scientific Literature | |
3. | ALA1041808 | Binding | Activity at human recombinant PTP1B | Homo sapiens | 8 | ALA1156122 | single protein format | Scientific Literature | |
4. | ALA1055397 | Functional | Inhibition of hypoxia-induced HIF1alpha protein accumulation in human Hep3B cells treated for 30 mins measured after 12 hrs by Western blot analysis | Homo sapiens | 16 | ALA1151479 | cell-based format | Scientific Literature | |
5. | ALA1055398 | Functional | Inhibition of hypoxia-induced VEGF protein secretion in human Hep3B cells after 16 hrs by ELISA | Homo sapiens | 16 | ALA1151479 | cell-based format | Scientific Literature | |
6. | ALA1056199 | Binding | Inhibition of hypoxia-induced HIF1 activation in human Hep3B cells by pGL3-HRE-luciferase reporter gene assay | Homo sapiens | 16 | ALA1151479 | cell-based format | Scientific Literature | |
7. | ALA995992 | Functional | Cytotoxicity against human HSC2 cells | Homo sapiens | 17 | ALA1155705 | cell-based format | Scientific Literature | |
8. | ALA995993 | Functional | Cytotoxicity against human HSG cells | Homo sapiens | 17 | ALA1155705 | cell-based format | Scientific Literature | |
9. | ALA995994 | ADME | Cytotoxicity against HGF cells | Homo sapiens | 17 | ALA1155705 | cell-based format | Scientific Literature | |
10. | ALA995995 | ADME | Selectivity ratio of CC50 for HGF cells to CC50 for human HSC2 cells | Homo sapiens | 16 | ALA1155705 | cell-based format | Scientific Literature |